* Canadian Radiation Protection Association Annual Conference (27/10/02)

Tramès per Klaus Rudolph (Citizens' Initiative Omega)

Dear Friends: In the month of Oct 1996 I found that I could no longer stay in my home which was located aprox.400 feet from a cellular phone tower, which also housed TV and FM radio antennae's.After living in that home for one year,and after intensive study,I found that the microwave emissions from the tower was the cause of my illness,which started soon after moving in ,pressure type headache,ringing ears,hearing loss,ear pain,fatigue,joint pain,blurry vision,sleep disturbance,sharp pain in sensitive areas, as well as other strange symptoms.

After several calls to the cellular phone company represetative that owned the antenna's,and not getting results, I threatened a law suit against them.The cellphone representative told me that he was sorry that I had said that because they were going to try to do something ,but they will now be taking a different approach to the situation. I mentioned to
the representative that I was having to sleep in a mobile home, that I had placed in my back yard, since the microwave emissions where unbearable inside my house. He then mentioned that he was by my home and had seen the mobile in my back yard.The next morning, ,just before dawn,while sleeping in my mobile,I was awakened by very high intensity
microwave.I ran to the door and jumped out of the mobile in my underwear,and I could hear a dog barking nearby.It was raining at the time.I held my hands tightly over my ears and after about half a minute the sound and pressure receded.I don't know how long this had gone on before I awakened, but It was so strong and load,that I felt like my
head was about to explode with pressure and it made me deaf for aprox 4 hours.

I waited until my doctors office opened and I drove directly there. When I stepped out of my van at the doctors office the sun was shining and I felt like I had been sunburnt on the left side of my neck and face. While I slept,I was lying on my right side with my feet toward the tower. The mobile was aprox.300 feet from the tower and there was a bush area
between the tower and my mobile. At least five people that saw me soon after,said that my left ear and neck area was bright red.Very noticeable. I had my doctor record the information.

The day this took place I received a phonecall ,at aprox 9 PM, from the cellphone representative telling me that they had been out to my place that morning, doing tests of the tower emissions ,and that they were sending the results to the Provincial Radiation Protection Officer ,and that they no longer want to hear from me.I would have to deal with the
PRPO.in the future. When I asked what happened in the early morning,he acted as though he didn't hear me,and kept
on talking.

Since that day I have become hypersensitive to microwave emissions and I suffer pain when ever I get near a tower or antenna.I had to sleep in a metal container for months after,in order to relieve the pain. The way the neighbors dog , that I heard barking in the morning of the event,a friendly white labrador, which I used to stop and pet from time to time,and which had been tied up for months in direct view of the tower, went mad,and had to be put down,not too long
after the event took place,(according to the owner.)

I have to believe that I was targeted.  I have no way of knowing who was responsible ,but I would be willing to swear an affidavit to the effect that the above information is true,and that the event did take place on a exact date which I have recorded.

Robert Riedlinger Mission B C Canada V4S 1H9

Canadian Radiation Protection Association Annual Conference Vancouver BC, May 6-9, 2002

Phosphorylation of Hsp27 - The Molecular Mechanism for Mobile Phone Radiation-Induced Increase in Blood-Brain Barrier Permeability.

Dariusz Leszczynski1,2, Sakari Joenväärä1, Jukka Reivinen1 and Reetta Kuokka1 The question of whether mobile phone radiation (RF-EMF) is hazardous to health remains unanswered. Earlier we have shown (Leszczynski et al., Differentiation, 70, 2002, in press) that the 1-hour non-thermal exposure of human endothelial cell line EA.hy926 to
SAR of 2W/kg (900MHz GSM signal) leads to: (i) transient increase in phosphorylation of hsp27 stress response protein, which was prevented by SB203580, a specific inhibitor of p38 mitogen-activated protein kinase (p38MAPK), and (ii) transient changes in protein expression levels of hsp27 and p38MAPK. Phosphorylation of hsp27 has been shown to regulate cell apoptosis - due to inhibition of the proteolytic activation of pro-caspase-9, and stability of stress fibers - due to increased polymerization of actin. The latter, when occurring in brain endothelial cells might affect functioning of blood-brain barrier. Thus, we have proposed that: the activation (phosphorylation) of hsp27 by mobile phone radiation might be the molecular mechanism regulating (i) caspase-9-dependent apoptotic pathway and (ii) increase in blood-brain barrier permeability, which has been observed in some animal experiments.

To determine whether physiological responses of endothelial cells, which are associated with the hsp27 phosphorylation and might affect permeability of blood-brain barrier (stability of stress fibers, cell size/shape), occur in the mobile phone radiation exposed cultures of human endothelial cells were executed experiments using cultures of
human endothelial cell line EA.hy926 cells, grown on microscope cover slides, were exposed for 1h to 900MHz GSM signal at an average SAR of 2W/kg. Temperature of cell cultures remained throughout irradiation period at 37±0.3°C thus the effects reported here are of non-thermal nature. Cells on cover slides were fixed either immediately or 1h after
the end of irradiation. The expression of hsp27 was determined by indirect immunohistochemistry. The appearance of cells (size, shape) and stabilization of stress fibers was determined by staining of the cells with phalloidin that was labeled with fluorescent-dye (AlexiaFluor).

As expected, 1h RF-EMF exposure increased expression of hsp27. However, in order to increase hsp27 expression by heat shock was required 3h incubation of cells at 43°C (1h exposure had no effect). This observation, together with the measurements showing that temperature of medium was throughout RF-EMF exposure period at 37±0.3°C, suggest that the observed here effects are of non-thermal nature. The stability of stress fibers, as determined by the pattern of staining with phalloidin-AlexiaFluor, increased after 1h irradiation and did not decline during the 1h of post-irradiation incubation The observed hsp27-related increase in the stability of stress fibers and caused by it changes in cell shape and size of EA.hy926 cells support the hypothesis of the regulatory role of hsp27/p38MAPK pathway RF-EMF-induced permeability of blood-brain barrier. Furthermore, the possible RF-EMF-induced breakage of the blood-brain barrier, if occurring repeatedly over a long period of time might become a health hazard because of the possible extra-capillary accumulation of molecules that might cause brain tissue damage.

1 Bio-NIR Research Group, Radiobiology Laboratory, Department of Research and Environmental Surveillance, STUK - Radiation and Nuclear Safety Authority, Helsinki, Finland
2 Presenting Author, dariusz.leszczynski@stuk.fi

Source: http://www.crpa-acrp.ca/CRPA_Papers/60.htm


The message is in reply to a message of Don Maisch:  
"How mobile phones let spies see our every move"

Alasdair Philips
Friday, October 25, 2002 12:05 PM
Subject: BAE involved in development of Celldar

[ This info dates from July; there is more info on Roke Manor's work at  http://www.roke.co.uk/news/article.asp?id=38

BAE SYSTEMS and Roke Manor Research Team to Develop Revolutionary Cellphone Radar Wednesday, July 24, 2002

BAE SYSTEMS and Roke Manor Research are teaming to develop in depth the concept of CELLphone raDAR - CELLDAR - to provide a revolutionary, totally covert and innovative approach to the detection of moving air, land and sea-based objects, maturing a technology which will significantly enhance military capabilities such as air warfare, littoral operations and Homeland Defence.

The two companies have signed an agreement to fund the development of the technology, already successfully developed by Roke Manor Research, exploiting the latter's in-depth knowledge of cellphone technology through its pivotal role within Siemens, a world leader in this market place, in enabling R&D. The BAE SYSTEMS Future Systems, C4ISR and other business units will be contributing their defence domain knowledge and systems integration expertise.

CELLDART uses extended multi-static radar detection and data processing for the tracking, identification and cueing of objects moving in cellphone fields. The massive world-wide investment in cellphone technology and the ability to exploit the extensive electromagnetic transmission fields created to support them presents the opportunity for CELLDART to offer high-performance, long-range, low-cost detection of objects moving in space in real time to user communities.

The capability of the technology extends across all domains and will be a key enabler in future Command & Control, Communications, Computing, Intelligence, Surveillance and Reconnaissance (C4ISR) solutions. It encompasses - for instance - the detection of moving vehicles or helicopters in foliage; of small maritime objects, such as periscopes; and even aircraft which would otherwise be invisible to traditional mono-static or bi-static radars through the exploitation of stealth technology.

CELLDART utilises the radar frequencies associated with the current mobile telephone transmissions (GSM 900, 1800 and 1900) and future transmissions (3G).

This partnership complements earlier initiatives to combine the skills of these two world-class companies. These resulted in the SAMPSON active phased array, multi-function radar (selected for the Royal Navy's Type 45 destroyer) and the HALO® artillery location system now in service with the British Army and being supplied into the export market.

Informant: Don Maisch

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